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glutathione apap Hepsin as a potential therapeutic target for alleviating acetaminophen-induced hepatotoxicity via gap-junction regulation and oxidative stress modulation | Cell Biology and Toxicology

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glutathione apap Hepsin as a potential therapeutic target for alleviating acetaminophen-induced  hepatotoxicity via gap-junction regulation and oxidative stress modulation  | Cell Biology and Toxicology

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glutathione apap Hepsin as a potential therapeutic target for alleviating acetaminophen-induced  hepatotoxicity via gap-junction regulation and oxidative stress modulation  | Cell Biology and Toxicology
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